INTEREST in the pathogenesis of peptic esophagitis has led to the concept that an incompetent intrinsic esophagogastric sphincter mechanism results in regurgitation of gastric secretion into the distal esophagus. Inflammation, ulceration, and subsequent healing with fibrosis and stricture formation occur. Resection of the obstructed area with interposition of a jejunal segment between the esophagus and stomach has been recommended by Merendino as a remedy for this difficult problem.1-6 The following is a case report of a patient who was treated in the above manner and who subsequently developed a high jejunal obstruction due to compression of the bowel by the taut interposed mesentery.
Report of Case
This 31-year-old white man was admitted to the Los Angeles County Harbor General Hospital on Nov 21, 1960, with the history of dysphagia, increased salivation, indigestion, and vomiting episodes since the age of four. He had an episode of severe hematemesis ten years