In the early days before the development of reconstructive operations for arteriosclerosis obliterans, many patients with vascular intermittent claudication came first to the orthopedist. The report in 1949 of Boyd et al1 did much to alert him to the need for looking beyond the skeletal system in such patients. Now, the passage of 27 years has made it necessary to alert vascular and general surgeons to the neurologic origin of some "claudication" symptoms. As the article by Karayannacos et al in this issue (p 803) states, vascular surgeons are frequently the first to see them.
Vascular surgeons see many patients with leg symptoms that are not vascular in origin, patients with faulty mechanics originating in foot, ankle, knee, hip, or back, patients with night cramps, patients with obesity (in whom the pulses are hard to feel), patients with asthenia and cold feet. We have therefore learned to be strict