RT Journal
A1 NOER RJ, DERR J
T1 REvascularization following experimental mesenteric vascular occlusion
JF Archives of Surgery
JO Archives of Surgery
YR 1949
FD May 1
VO 58
IS 5
SP 576
OP 589
DO 10.1001/archsurg.1949.01240030586002
UL http://dx.doi.org/10.1001/archsurg.1949.01240030586002
AB THE SMALL intestine has a remarkable ability to withstand interruption of its blood supply. The distribution and anastomoses of its vessels appear to be responsible for its great revascularizing potential, and it has been generally assumed that the peculiar arcuate system of the intestinal tract is the chief factor in overcoming vascular deprivations. Considerable experimental effort has been devoted to determining the degree of vascular interruption which is compatible with life, yet the exact mode by which compensation takes place has not been clearly established. The literature pertinent to these studies may be discussed under two categories—(1) anatomic studies and (2) experimental mesenteric vascular interruption and revascularization of the intestine.ANATOMIC STUDIES 
    Morphologic reports on the larger vessels of the small intestine are in accord, but many discrepancies appear in descriptions of the vasa recta and the vessels within the intestinal wall. Much of the confusion doubtless arises from the